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Lattice degeneration is a disease of the human eye wherein the peripheral retina becomes atrophic in a lattice pattern. Usually, this happens slowly over time and does not cause any symptoms, and medical intervention is neither needed nor recommended.
Retinal tufts can be visualized or diagnosed using a dilated eye examination and indirect ophthalmoscope or a widefield retinal scan. [3] A retinal tuft is a gliotic degeneration of the retina composed of focal adhesions in the extracellular matrix joining the retina and the posterior hyaloid of the eye. [4] [5]
Retinoschisis is an eye disease characterized by the abnormal splitting of the retina's neurosensory layers, usually in the outer plexiform layer.Retinoschisis can be divided into degenerative forms which are very common and almost exclusively involve the peripheral retina and hereditary forms which are rare and involve the central retina and sometimes the peripheral retina.
The risk of retinal detachment is the greatest in the first 6 weeks following a vitreous detachment, but can occur over 3 months after the event.. The risk of retinal tears and detachment associated with vitreous detachment is higher in patients with myopic retinal degeneration, lattice degeneration, and a familial or personal history of previous retinal tears/detachment.
Lattice degeneration: Thinning of the retina, which increases its susceptibility to breaks or tears. [ 9 ] [ 10 ] [ 18 ] Cystic retinal tuft : A small, raised spot present on the retina from birth that increases the risk for tears and detachment.
Fundus photographs are also used to document abnormalities of disease process affecting the eye, and/or to follow up on the progress of the eye condition/disease such as diabetes, age-macular degeneration (AMD), glaucoma, multiple sclerosis, and neoplasm of the choroid, cranial nerves, retinal or eyeball.
It has no systemic manifestations, unlike the other type of the dystrophy, Lattice corneal dystrophy type II. Lattice corneal dystrophy was first described by Swiss ophthalmologist Hugo Biber in 1890. [1] Lattice dystrophy gets its name from an accumulation of amyloid deposits, or abnormal protein fibers, throughout the middle and anterior stroma.
Defects in this gene are associated with both central and peripheral retinal degenerations. Some of the various phenotypically different disorders are autosomal dominant retinitis pigmentosa, progressive macular degeneration, macular dystrophy and retinitis pigmentosa digenic. [6]
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