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The Wasserman 9-Panel Plot, often called a Nine-Panel Plot, is a standard layout for the graphical representation of data produced by a cardiopulmonary exercise test. [1] The layout was updated in 2012. The graphs give an overview of cardiovascular, ventilatory, and gas exchange parameters. [2]
Pulmonary function testing is a safe procedure; however, there is cause for concern regarding untoward reactions and the value of the test data should be weighed against potential hazards. Some complications include dizziness, shortness of breath, coughing, pneumothorax, and inducing an asthma attack.
The deep part of the cardiac plexus is situated in front of the bifurcation of the trachea (known as the carina), above the point of division of the pulmonary artery, and behind the aortic arch. It is formed by the cardiac nerves derived from the cervical ganglia of the sympathetic trunk, and the cardiac branches of the vagus and recurrent ...
Place the bony parts of the palm around the borders of the patient's scapulae while he or she says "ninety-nine" or "one one one" to test for fremitus. Repeat the sequence on the front of the chest. A decrease in fremitus may be observed if the patient has a soft voice, obstructed bronchus, COPD , pneumothorax , or other disease or injuries ...
Cardiac stress testing is used to determine to assess cardiac function and to disclose evidence of exertion-related cardiac hypoxia. Radionuclide testing using thallium or technetium can be used to demonstrate areas of perfusion abnormalities. With a maximal stress test the level of exercise is increased until the person's heart rate will not ...
Cardiac physiology or heart function is the study of healthy, unimpaired function of the heart: involving blood flow; myocardium structure; the electrical conduction system of the heart; the cardiac cycle and cardiac output and how these interact and depend on one another.
cardiac causes, pulmonary causes and; non-pulmonary and non-cardiac causes. Considered physiologically, pulsus paradoxus is caused by: [citation needed] decreased right heart functional reserve, e.g. myocardial infarction and tamponade, right ventricular inflow or outflow obstruction, e.g. superior vena cava obstruction and pulmonary embolism, and
The pathophysiology of pulmonary heart disease (cor pulmonale) has always indicated that an increase in right ventricular afterload causes RV failure (pulmonary vasoconstriction, anatomic disruption/pulmonary vascular bed and increased blood viscosity are usually involved [1]), however most of the time, the right ventricle adjusts to an overload in chronic pressure.