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Gilbert syndrome (GS) is a syndrome in which the liver of affected individuals processes bilirubin more slowly than the majority. [1] Many people never have symptoms. [ 1 ] Occasionally jaundice (a slight yellowish color of the skin or whites of the eyes) may occur.
Gilbert Syndrome, a hereditary disease affecting 5% of the US population, leads to stress-induced mild hyperbilirubinemia. [9] The mutated UGT1 gene produces defective UGT1A1 enzymes vital for bilirubin conjugation.
Gilbert's syndrome, the Cleveland Clinic explains, “affects the liver’s ability to process bilirubin,” which is produced by the breakdown of red blood cells and makes those with Gilbert's ...
Gilbert Syndrome is a partial reduction in UGT1A1's activity which causes transient hyperbilirubinemia during various stressors, like fasting or acute illness. Gilbert syndrome is inherited in both autosomal dominant and autosomal recession nature. Crigler-Najjar syndrome is much more severe and presents in newborns.
According to the Mayo Clinic, Gilbert syndrome is a common and harmless liver condition caused by a modified gene inherited from parents.
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Gilbert's syndrome – a genetic disorder of bilirubin metabolism that can result in mild jaundice, found in about 5% of the population; Rotor syndrome: non-itching jaundice, with rise of bilirubin in the patient's serum, mainly of the conjugated type; Dubin–Johnson syndrome; Crigler–Najjar syndrome
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