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In NICO the pain is said to result from the degenerating nerve ("neuralagia"). [2] [3] [1] The condition is probably rare, if it does exist. [4] Also called Ratner's bone cavity, a neuralgia-inducing cavitational osteonecrosis was first described in dental literature by G V Black in 1920. [5]
Tooth extraction is the major risk factor for development of MRONJ. Prevention including the maintenance of good oral hygiene, comprehensive dental examination and dental treatment including extraction of teeth of poor prognosis and dentoalveolar surgery should completed prior to commencing any medication which is likely to cause osteonecrosis ...
Periapical granuloma, [1] also sometimes referred to as a radicular granuloma or apical granuloma, is an inflammation at the tip of a dead (nonvital) tooth. It is a lesion or mass that typically starts out as an epithelial lined cyst, and undergoes an inward curvature that results in inflammation of granulation tissue at the root tips of a dead tooth.
The signs and symptoms depend upon the type of OM, and may include: Pain, which is severe, throbbing and deep-seated and often radiates along the nerve pathways. Initially fistula are not present. Headache or facial pain, as in the descriptive former term "neuralgia-inducing" (cavitational osteonecrosis). Fibromyalgia. Chronic fatigue syndrome ...
For example, the incidence of nerve injury in teens removing third molars is much lower than the incidence in patients 25 and older. [7] This risk increases 10 fold if the tooth is close to the inferior dental canal containing the inferior alveolar nerve (as judged on a dental radiograph). [8]
Initial stage: Epithelial cells from the rests of Malassez at the apex of the roots of a non-vital tooth (one where the nerve and blood supply in the tooth have degenerated and no longer exist) become stimulated due to the body's inflammatory response to bacterial endotoxins infecting the pulp or as a direct response to necrotic pulp tissue ...
Condensing osteitis happens when the bone around the tooth reacts to long-term inflammation. This involves excessive bone growth, leading to the formation of sclerotic bone in the jaw. Ongoing tooth infections, like pulpitis, release chemicals that attract immune cells and activate osteoblasts. Osteoblasts create extra bone in response to ...
As the dental pulp is a highly vascularised and innervated region of the tooth, it is the site of origin for most pain-related sensations. [12] The dental pulp nerve is innervated by one of the trigeminal nerves, otherwise known as the fifth cranial nerve. The neurons enter the pulp cavity through the apical foramen and branch off to form the ...