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Treatment of childhood dementia focuses on managing symptoms and improving quality of life. This can include: Medications: Anti-seizure medications, behaviour-modifying drugs, and muscle relaxants. [1] Therapies: Physiotherapy, occupational therapy, and speech therapy are used to maintain physical function for as long as possible. [1]
The symptoms must not be the result of a mechanical obstruction, and should not respond to the standard treatment for gastroesophageal reflux disease. [ 2 ] In adults, the diagnosis is supported by the absence of classical or structural diseases of the gastrointestinal system.
Mild cognitive impairment (MCI) is a diagnosis that reflects an intermediate stage of cognitive impairment that is often, but not always, a transitional phase from cognitive changes in normal aging to those typically found in dementia, [1] especially dementia due to Alzheimer's disease (Alzheimer's dementia). [2]
Dementia and delirium are the cause of the confusion, orientation, cognition or alertness impairment. [11] Therefore, these symptoms require more attention because hallucinations, delusions, amnesia, and personality changes are the result. These effects of the dementia and delirium are not joined with the changes of sensory or perception abilities.
The treatment will depend on the cause of memory loss, but various drugs to treat Alzheimer's disease have been suggested in recent years. There are four drugs currently approved by the Food and Drug Administration (FDA) for the treatment of Alzheimer's, and they all act on the cholinergic system: Donepezil , Galantamine , Rivastigmine , and ...
Primary familial brain calcification [1] (PFBC), also known as familial idiopathic basal ganglia calcification (FIBGC) and Fahr's disease, [1] is a rare, [2] genetically dominant or recessive, inherited neurological disorder characterized by abnormal deposits of calcium in areas of the brain that control movement.
The hallmark symptom of LATE is a progressive memory loss that predominantly affects short-term and episodic memory. [1] This impairment is often severe enough to interfere with daily functioning and usually remains the chief neurologic deficit, unlike other types of dementia in which non-memory cognitive domains and behavioral changes might be noted earlier or more prominently. [1]
Signs and symptoms are classified into three groups based on the affected functions of the frontal and temporal lobes: [8] These are behavioural variant frontotemporal dementia, semantic dementia, and progressive nonfluent aphasia. An overlap between symptoms can occur as the disease progresses and spreads through the brain regions. [14]
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