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Ocular hypertension is the presence of elevated fluid pressure inside the eye (intraocular pressure), usually with no optic nerve damage or visual field loss. [1] [2]For most individuals, the normal range of intraocular pressure is between 10 mmHg and 21 mmHg. [3]
Intraocular pressure (IOP) is the fluid pressure inside the eye. Tonometry is the method eye care professionals use to determine this. IOP is an important aspect in the evaluation of patients at risk of glaucoma. [1] Most tonometers are calibrated to measure pressure in millimeters of mercury .
For eye pressures, a value of 28 hPa (21 mmHg) above atmospheric pressure 1,010 hPa (760 mmHg) is often used, with higher pressures leading to a greater risk. [2] [26] However, some may have high eye pressure for years and never develop damage. [2] Conversely, optic nerve damage may occur with normal pressure, known as normal-tension glaucoma. [27]
Hypotony has many causes including post-surgical wound leak from the eye, chronic inflammation within the eye including iridocyclitis, hypoperfusion, tractional ciliary body detachment or retinal detachment. [5] Eye inflammation, medications including anti glaucoma drugs, or proliferative vitreoretinopathy causes decreased production. [6]
Oxygen toxicity is a condition resulting from the harmful effects of breathing molecular oxygen (O 2) at increased partial pressures.Severe cases can result in cell damage and death, with effects most often seen in the central nervous system, lungs, and eyes.
Eye-gouging is the act of pressing or tearing the eye using the fingers or instruments. Eye-gouging involves a very high risk of eye injury , such as eye loss or blindness. Eye-gouging as a fighting style was once a popular form of sport fighting in the back-country United States , primarily in the 18th and 19th centuries.
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Signs of damage to the retina caused by hypertension include: [citation needed]. Laser Doppler imaging of the papilla of a patient with hypertension. Arteriolar changes, such as generalized arteriolar narrowing, focal arteriolar narrowing, arteriovenous nicking, changes in the arteriolar wall (arteriosclerosis) and abnormalities at points where arterioles and venules cross.
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