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PRL-8-53 is a nootropic substituted phenethylamine that has been shown to act as a hypermnesic drug in humans; it was first synthesized by medical chemistry professor Nikolaus Hansl at Creighton University in the 1970s as part of his work on amino ethyl meta benzoic acid esters.
During the process, six liters (1.585 gallons) of hydrochloride of zinc and aluminum was injected into his common carotid artery. [2] In 1912, Brazilian educator and archivist Luís Gastão d'Escragnolle Dória went to the emperor's glass-topped coffin. At the occasion, he had noticed the well-preserved state of Pedro II's body. [3]
1-Ethyl-3-(3-dimethylaminopropyl)carbodiimide (EDC, EDAC or EDCI) is a water-soluble carbodiimide usually handled as the hydrochloride. [1] It is typically employed in the 4.0-6.0 pH range. It is generally used as a carboxyl activating agent for the coupling of primary amines to yield amide bonds.
Triethylenetetramine (TETA and trien), also known as trientine when used medically, is an organic compound with the formula [CH 2 NHCH 2 CH 2 NH 2] 2.The pure free base is a colorless oily liquid, but, like many amines, older samples assume a yellowish color due to impurities resulting from air oxidation.
Etilefrine, also known as 3,β-dihydroxy-N-ethylphenethylamine, is a substituted phenethylamine derivative. [2]It is an analogue of epinephrine (3,4,β-trihydroxy-N-methylphenethylamine), of phenylephrine ((R)-β,3-dihydroxy-N-methylphenethylamine), of metaterol (3,β-dihydroxy-N-isopropylphenethylamine), and of norfenefrine (3,β-dihydroxyphenethylamine), as well as of metaraminol ((1R,2S)-3 ...
β-Alanine ethyl ester is the ethyl ester which hydrolyses within the body to form β-alanine. [4] It is produced industrially by the reaction of ammonia with β-propiolactone. [5] Sources for β-alanine includes pyrimidine catabolism of cytosine and uracil.
The acid 1 will react with the carbodiimide to produce the key intermediate: the O-acylisourea 2, which can be viewed as a carboxylic ester with an activated leaving group. The O-acylisourea will react with amines to give the desired amide 3 and urea 4. The possible reactions of the O-acylisourea 2 produce both desired and undesired products.
[3] After instillation of cyclopentolate, pupil dilation ( mydriasis ) typically lasts up to 24 hours, while paralysis of the ciliary muscle ( cycloplegia ) typically lasts 6-24 hours. [ 4 ] During this time, patients may be more light sensitive than normal and may notice close objects blurred (and possibly distant objects blurred, depending on ...