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The metabolic stability theory of aging suggests it is the cells ability to maintain stable concentration of ROS which is the primary determinant of lifespan. [37] This theory criticizes the free radical theory because it ignores that ROS are specific signalling molecules which are necessary for maintaining normal cell functions. [37]
It is predicted that the knowledge of methods to increase the length of cell telomeres (Stem cell and quasi-stem cells, control the regeneration and rebuilding of different tissues of the body) will pave the way for increasing human lifespan. [10] [11] Examining telomeres is one of the most important fields of research related to aging.
By 2050, global life expectancy, which was 72.6 years in 2019, is expected to increase by approximately five years. [166] Maintaining good nutritional status and adequate nutrient intake is essential for health, quality of life, and overall well-being in older age, and it plays a crucial role in healthy aging as defined by the WHO. [168]
Higher levels of omega-6 fatty acids often found in ultraprocessed foods may interfere with the immune system’s fight against cancer cells, a new study says.
Put the phone down and grab a book. Whether you prefer a steamy romance novel, a fascinating piece of nonfiction, or a moody mystery, reading boasts tons of benefits for your brain.
Senescence (/ s ɪ ˈ n ɛ s ə n s /) or biological aging is the gradual deterioration of functional characteristics in living organisms. Whole organism senescence involves an increase in death rates or a decrease in fecundity with increasing age, at least in the later part of an organism's life cycle.
Embryonic stem cells and germ cells have also been described as immortal. [8] [9] Immortal cell lines of cancer cells can be created by induction of oncogenes or loss of tumor suppressor genes. One way to induce immortality is through viral-mediated induction of the large T-antigen, [10] commonly introduced through simian virus 40 (SV-40). [11]
DNA damage causes the cells to stop dividing or induces apoptosis, often affecting stem cell pools and therefore hindering regeneration. However, lifelong studies of mice suggest that most mutations happen during embryonic and childhood development, when cells divide often, as each cell division is a chance for errors in DNA replication. [ 88 ]