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In July 2000, docosanol was approved for medical use in the United States as an antiviral agent for reducing the duration of cold sores. [3] [4] [7] It is an over-the-counter medication (OTC). It is sold under the brand name Abreva among others. [3] [5] [8] [9]
List of Antiviral Drugs Antiviral Use Manufacturer Component Type Year approved Abacavir: HIV: ViiV Healthcare: Nucleoside analogue reverse transcriptase inhibitor (NRTI) 1998 Acyclovir (Aciclovir) Herpes Simplex, chickenpox, [2] varicella zoster virus: GSK: guanosine analogue RTI 1981 Adefovir: Hepatitis B [3] Gilead Sciences RTI 2002 , 2003 ...
Prescription antiviral medications slow down viral replication so sores heal faster, but no drug can eliminate the virus entirely. “We simply cannot figure out how to get rid of that latent ...
Antiviral creams, including over-the-counter Abreva (docosanol cream), are typically the first line of defense when you develop a cold sore on your lip, he says. These creams may heal your cold ...
Famciclovir is a guanosine analogue antiviral drug used for the treatment of various herpesvirus infections, most commonly for herpes zoster (shingles). It is a prodrug form of penciclovir with improved oral bioavailability. Famciclovir is marketed under the trade name Famvir .
Cold sore; Other names: fever blister, [1] herpes labialis, [1] oral herpes, [2] orolabial herpes [3] A cold sore on the lower lip. Note the blisters in a group marked by an arrow. Specialty: Infectious disease: Symptoms: Burning pain followed by small blisters or sores [1] Complications: Herpes encephalitis, herpetic whitlow [4] Usual onset ...
Because penciclovir is absorbed poorly when given orally (by mouth) it is more often used as a topical treatment. It is the active ingredient in the cold sore medications Denavir (NDC 0135-0315-52), Vectavir and Fenivir. Famciclovir is a prodrug of penciclovir with improved oral bioavailability. Penciclovir was approved for medical use in 1996. [2]
If a virus is not fully wiped out during a regimen of antivirals, treatment creates a bottleneck in the viral population that selects for resistance, and there is a chance that a resistant strain may repopulate the host. [53] Viral treatment mechanisms must therefore account for the selection of resistant viruses.
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