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Cervical cancer is the 12th-most common cancer in women in the UK (around 3,100 women were diagnosed with the disease in 2011), and accounts for 1% of cancer deaths (around 920 died in 2012). [148] With a 42% reduction from 1988 to 1997, the NHS-implemented screening programme has been highly successful, screening the highest-risk age group (25 ...
[1] [2] HeLa cells are durable and prolific, allowing for extensive applications in scientific study. [3] [4] The line is derived from cervical cancer cells taken on February 8, 1951, [5] from Henrietta Lacks, a 31-year-old African American mother of five, after whom the line is named. Lacks died of cancer on October 4, 1951.
Henrietta Lacks (born Loretta Pleasant; August 1, 1920 – October 4, 1951) [2] was an African-American woman [5] whose cancer cells are the source of the HeLa cell line, the first immortalized human cell line [B] and one of the most important cell lines in medical research.
This work was followed by a larger trial testing HPV screening – the HART study. Published in 2003, evidence from this played a role in the decision of the International Agency for Research on Cancer (IARC) to recommended that HPV testing could be used in primary cervical screening in 2004. [3]
Karen Canfell is an Australian epidemiologist and cancer researcher.. After being awarded a D.Phil. from Oxford in 2004 with a thesis entitled Use of hormone replacement therapy as a potential co-factor in the neoplastic progression of HPV-related cervical disease, [1] Canfell returned to Australia to work for the Cancer Council in Sydney where she continued to work on cervical cancer in ...
Bradley J. Monk is an American gynecologic oncologist, academician and researcher. He is a professor on the Clinical Scholar Track in the Department of Obstetrics and Gynecology at the University of Arizona College of Medicine in Phoenix, Arizona, [1] as well as at the Creighton University School of Medicine in Omaha, Nebraska.
Microplastics that have been found in human organs have “alarming links” to adverse health impacts, including lesions, cervical cancer, and other diseases, researchers suggest. Now, an ...
The cause of CIN is chronic infection of the cervix with HPV, especially infection with high-risk HPV types 16 or 18. It is thought that the high-risk HPV infections have the ability to inactivate tumor suppressor genes such as the p53 gene and the RB gene, thus allowing the infected cells to grow unchecked and accumulate successive mutations, eventually leading to cancer.
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