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2. Remyelination - Wikipedia

   en.wikipedia.org/wiki/Remyelination

   The processes underlying remyelination are under investigation in the hope of finding treatments for demyelinating diseases, such as multiple sclerosis. As of 2022 the status of possible remyelination acceleration is of trials only, [2] with side effects of possible drugs one limiting issue. [3]

3. Management of multiple sclerosis - Wikipedia

   en.wikipedia.org/wiki/Management_of_multiple...

   [16] [40] In 2013 one further oral drug, dimethyl fumarate -or BG12- (which is an improved version of fumaric acid, an already existing drug), was approved by the FDA. Another oral drug, cladribine, was approved in Russia and Australia in 2010. Its application was rejected by the FDA and EMEA in 2011, due to safety concerns.

4. Cerebrolysin - Wikipedia

   en.wikipedia.org/wiki/Cerebrolysin

   Cerebrolysin (developmental code name FPF-1070) is an experimental mixture of enzymatically-treated peptides derived from pig brain whose constituents can include brain-derived neurotrophic factor (BDNF), glial cell line-derived neurotrophic factor (GDNF), nerve growth factor (NGF), and ciliary neurotrophic factor (CNTF).

5. New treatment may stop and potentially reverse some nerve ...

   www.aol.com/treatment-may-stop-potentially...

   Multiple sclerosis (MS) is a chronic disease of the nervous system that can lead to muscle weakness, vision loss, and paralysis. It occurs when the immune system attacks the myelin sheath that ...

6. Multiple sclerosis drug pipeline - Wikipedia

   en.wikipedia.org/wiki/Multiple_sclerosis_drug...

   hydralazine: Due to its ability to damage myelin nerve sheaths, acrolein may be a factor in the development of multiple sclerosis. The antihypertensive drug hydralazine, a known scavenger of acrolein, was found to reduce myelin damage and significantly improve behavioral outcomes in a mouse model of multiple sclerosis (experimental autoimmune ...

7. Lesional demyelinations of the central nervous system

   en.wikipedia.org/wiki/Lesional_demyelinations_of...

   A repair process, called remyelination, takes place in early phases of the disease, but the oligodendrocytes are unable to completely rebuild the cell's myelin sheath. Repeated attacks lead to successively less effective remyelinations, until a scar-like plaque is built up around the damaged axons.