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The primary causes include post-cataract surgery, certain medications, and, less commonly, neurological or ophthalmological conditions. Post-cataract surgery is a common cause, as replacing the natural lens with a synthetic one increases exposure to blue light, leading to temporary blue-tinted vision. This effect usually resolves as the eye adapts.
Visual outcome after cataract surgery was close to conformity with WHO guidelines in Buenos Aires, where more than 80% of post-surgery eyes had visual acuity of 6/18 (20/60) or better, but ranged between 60% and 79% in most of the other regions, and was less than 60% in Guatemala and Peru. [126]
Pressure phosphenes can persist briefly after the rubbing stops and the eyes are opened, allowing the phosphenes to be seen on the visual scene. Hermann von Helmholtz and others have published drawings of their pressure phosphenes. One example of a pressure phosphene is demonstrated by gently pressing the side of one's eye and observing a ...
Posterior capsular opacification, also known as after-cataract, is a condition in which months or years after successful cataract surgery, vision deteriorates or problems with glare and light scattering recur, usually due to thickening of the back or posterior capsule surrounding the implanted lens, so-called 'posterior lens capsule opacification'.
After cataract surgery, patients with diabetes mellitus are generally acknowledged to have an increased risk of macular edema. [12] A prior history of retinal vein occlusion was the only significant preoperative risk factor in a large retrospective series of 1659 consecutive cataract surgeries. [13]
One drop of xylocaine instilled four times after every 4 minutes will produce conjunctival and corneal anaesthesia. Paracaine, tetracaine, bupivacaine, lidocaine etc. may also be used in place of xylocaine. [1] Cataract surgery by phacoemulsification is frequently performed under surface anaesthesia.
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